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00二、论坛地点华南理工大学大学城校区B2-513会议

来源:http://www.qd-haiyu.com 作者:澳门金莎娱乐网站-官方首页 时间:2019-09-13 11:59

二零一八年中华夏族民共和国海洋大学国外青少年学者“越崎论坛”

常见师生: 华工“海内外卓绝青少年学者论坛”目的在于面向全世界邀约具备不一样学术背景的青少年才俊,围绕国际科学前沿、火爆商量世界以及行当行当的才干难点等举办研商和沟通。希望藉此平台,相互启发、开采视线,巩固国际交换与合营,促进两岸联袂前进。现将经济高校分论坛有关陈设公告如下:一、论坛时间二零一八年10月20日14:00-17:00二、论坛地方华法高校城校区B2-513开会地点三、论坛章程

周围师生:华工“海内外优良青年学者论坛”意在面向全世界特邀青少年才俊,围绕国际科学战线、热门研商世界以及行当行业的技术难点等开展斟酌和调换。籍此平台,启迪观念,开发视线,推动学术调换与搭档,此番论坛的具体安顿如下:一、论坛时间二零一八年7月8日清晨15:00-16:00二、论坛地方华工4号楼数学大学4318会议厅三、论坛章程1. 15:00-15:05高校监护人致招待辞;2. 15:05-16:00报告人:沙敏大学生,澳洲McCaw瑞大学,报告标题:代数数的乘性相关性(Multiplicative dependence of algebraic numbers)。应接广大师生到场!数学大学2018年8月二十四日内容摘要:代数数的乘性相关性是数论中的特出课题。大家称n个非零复数a1, ..., an是乘性相关的,假若存在不全为零的整数k1, ..., kn使得乘积a1k1... ankn=1。同理,称二个n维向量是乘性相关的,假设它的n个坐标是乘性相关的。报告将从分析、算术和几何角度开展。从解析角度,思索到稠密性难点。在实空间或复空间中,就算乘性相关向量的勒贝格估摸为零,但注脚它们在空中中是黑压压的,並且向量的坐标只需取自有个别代数数域。然后通过考虑覆盖半径,这些标题能够被更周详地商讨。从算术角度,思量到n维乘性相关向量的计数难点。比方,假诺n维乘性相关向量的坐标是代数数,次数和冲天皆有上界,那么对于那一个向量的个数,给出了关于中度的渐近公式。从几何角度,记挂到代数曲线上的乘性相关点。注明即使曲线定义在一个代数数域K上而且亏格大于零,那么曲线上独有有限个乘性相关点,其坐标来自于K的天崩地坼Abe尓扩充。亏格为零的曲线也是有左近结论。报告人简单介绍:沙敏,一九八二年7月出生,研究生。二零零五年在华工获得博士学位,二〇〇八年在清华东军大学获得硕士学位,二〇一三年在法兰西福州高校得到大学生学位。二零一一年七月至2014年4月在澳洲新南Will士大学做硕士后。二〇一六年5月迄今在澳大利亚联邦(Commonwealth of Australia)McCaw瑞高校做校级大学生后。沙敏大学生短期研商数论及其使用。近些日子关键探讨兴趣包涵代数数论,椭圆曲线,有限域理论,算术重力系统,线性递归连串,以及数论中的图论难点。在包蕴Transactions of the American Mathematical Society, International Mathematics Research Notices, Moscow Mathematical Journal, Journal of Combinatorial Theory Series B等国际名牌杂志上刊登故事集多篇。从二零一二年起出任美利坚合资国《数学斟酌》的商量员。于二〇一一年获得欧洲缔盟委员会的Alain Bensoussan Fellowship,并于2014年获得McCaw瑞大学的Macquarie University Research Fellowship。

大规模师生:

能源高校分论坛

日期

附件:

华工“海内外优良青少年学者论坛”碰着与财富高校分论坛目的在于面向满世界约请具备分化学术背景的青少年才俊,围绕国际科学前沿、抢手钻探世界以及行当行业的技术难题等开展斟酌和调换。通过那一个平台,相互启发、开荒视界,巩固国际交换与搭档,推动双方共同前进。现将情状与财富高校分论坛有关布置通告如下:

间:2018年6月6日-7日

时间

一、论坛时间

主办单位:财富与地学高校

须知或章程

2017年12月25日上午9:15-12:30

接待全校师生踊跃插手!

地点

二、论坛地方

率先开会地点

10月23日

条件与能源大学B4-308开会地点

地址:煤层气能源与成藏进度教育部最主要实验室319开会地点

全天

三、论坛章程

(文昌校区西门外高校科技(science and technology)园B座)

报到、入住;

时间

时间:2018年6月7日9点

高档高校城宗旨饭馆

须知或章程

报告一:Particle-based modeling of pull-apart basin development

10月24日

9:15-9:30 开幕式

报告人:刘源,博士后, 德意志联邦共和国Frye贝格财经大学

14:00-17:00学术报告主持人:廉哲雄

高校总管致迎接词

告知摘要:

报告人:曾筑天(加拿大圣Jose高校Cumming 文大学博士后)标题:To See the Unseen: Intravital imaging reveals key immune defense mechanisms against bloodstream bacterial infection

9:30-12:30

1. A scale-independent modeling approach based on the Discrete Element Method has been built to investigate pull-apart basin development.

B2-513会议室

学术报告

2. The shape of a pull-apart basin is the consequence of both initial strike-slip geometry and its various evolution stages.

报告人:叶浩彬(佛罗里达高校历史高校硕士后)标题:Management of Leukemia from Metabolic Perspectives

主题材料:大气污染物的遥感观测本事(Remote sensing observations of atmospheric aerosols and trace 瓦斯es)

3. Minimum displacements to form pull-apart basins, and minimum ages of initiation for pull-apart basins can be estimated.

招待广大师生参与! 历史大学二零一八年5月十26日1.报告人:曾筑天(加拿大圣多明各高校Cumming 管理大学大学生后)报告标题:To See the Unseen: Intravital imaging reveals key immune defense mechanisms against bloodstream bacterial infection内容摘要:Bloodstream bacterial infection is on the rise due to the wide spread use of indwelling intravenous catheters and immunosuppressive iatrogenic interventions. First-pass clearance of blood-borne bacteria is critical to control bloodstream infections and to prevent systemic dissemination. The liver has been well known as a blood-filtering organ capable of sequestering circulating bacteria via its vast pool of intravascular macrophages-Kupffer cells. However, the molecular mechanism underlying Kupffer cell mediated capture of circulating pathogens under shear conditions remains less understood. 塔基ng advantage of high-speed real time intravital imaging, we visualized the dynamic process of bacterial capture by Kupffer cells, and revealed novel mechanisms utilized by Kupffer cells to catch bacteria. We observed a pattern recognition role for complement receptor-C凯雷德Ig in the capture of circulating Gram-positive bacteria from the bloodstream by directly binding to the Lipoteichoic Acid of Gram-positive bacteria, such as S. aureus. We also observed a sex-biased difference of Kupffer cells in capture circulating enteropathogenic E. coli . While complement opsonization was indispensable for the capture of EPEC in male mice; however, a faster, complement-independent process involving abundant preexisting antibodies to EPEC was detected in female mice. These antibodies were elicited predominantly in female mice at puberty in response to estrogen regardless of microbiota-colonization conditions. Estrogen-driven antibodies were maternally transferrable to offspring and conferred protection during infancy. Thus, an estrogen-driven, innate antibody-mediated immunological strategy conferred protection to females and their offspring. 报告人简要介绍︰曾筑天大学生于二〇〇〇年至2009年就读于科大生命科学大学,获得博士学位。随后在中国科学本事高校免疫性学切磋所师从田志刚院士并于二零一四年八月获取细胞生物学大学生学位。二〇一六年11月于今,曾筑天大学生在加拿大西雅图大学师从国际名牌免疫学专家PaulKubes讲师举办学士后商讨。在大学生及学士后斟酌时期,曾筑天大学生在肝脏天然免疫性学及感染应答机制等偏向做出一文山会海具备可持续性和创新型的关键研商,重要运用高分辨率活体显微成像本领完毕了对肝脏局地免疫性细胞和血液中微型生物动态相互作用的实时成像观测,揭破了肝脏抗细菌感染免疫性的成员细胞机制,开掘了斩新的由雌激素诱导的自发抗体新群众体育,并对慢性传播病魔毒感染状态下肝脏免疫机能低下致使乙型病毒性肝性传播病痛毒不可能被有效解决注明了机制。其研商成果以率先作者身份公布在了Nature Immunology, Cell Host&Microbe, Journal of Experimental Medicine, Journal of Immunology等高质量免疫性学期刊上。曾筑天大学生是免疫学领域的特出青少年学者,多年来致力于肝脏病魔免疫性学科学研讨及潜在免疫性医治格局的付出,在肝脏免疫性这一重中之重领域有所深厚的钻研功底,具备不可磨灭合理的前途计划以及伟大的发展潜质。曾筑天博士精晓卓殊的实验商讨手艺,能对活体小动物肝脏,肾脏,脾脏等八个脏器免疫细胞实行直接实时动态的显微成像检查评定及解析,是当前国际上为数非常的少的能对上述七个脏器进行活体动态成像商讨的大方之一。 2.报告人:叶浩彬(伊利诺伊大教育水平史大学大学生后)报告标题:Management of Leukemia from Metabolic Perspectives内容摘要:Obese leukemia patients have a poorer prognosis compared to normal weight patients, suggesting that obesity-associated conditions protect leukemia cells/leukemia stem cells from chemotherapy. Further, obese population have a higher risk for leukemia, indicating that obesity promotes disease development and progression. However, the biological mechanisms underlying these phenomena remain unknown. In today’s presentation, the author introduces two studies that reveal the mechanisms for the phenomena mentioned above. The first study has demonstrated that adipose tissue functions as a sanctuary for LSCs. Briefly, LSCs are found to be enriched in adipose tissue. Tranome comparisons show that compared to hematopoietic tissue-resident LSCs, adipose-resident LSCs display a pro-inflammatory gene signature, which leads to an inflamed state in adipose tissue, and consequently an increased lipolysis rate as evidenced by elevated serum fatty acids level. Lipolysis-derived fatty acids are utilized by two distinct pathways: 1) fatty acids-induced inflammation pathway and 2) fatty acid oxidation pathway. Interestingly, a LSC subpopulation that has a high-level expression of the fatty acid transporter CD36 (CD36+ LSCs) displays a significantly higher FAO rate compared to CD36- LSCs and is strikingly enriched in adipose tissue. Further, CD36+ LSCs are more chemo-resistant compared to CD36- LSCs partially due to CD36-mediated FAO. More importantly, a CD36+ LSC subpopulation is also observed in primary human leukemia patient samples. Human CD36+ LSCs display a higher FAO rate and are chemo-resistant compared to CD36- LSCs. Collectively, this study shows that the interplay between leukemia cells and adipose tissue creates a unique microenvironment that supports the metabolic demands and survival of a distinct LSC population.The second study demonstrates that leukemic disease causes aberrancies in multiple tissues including adipose tissue, pancreas, gut and gut microbiota to subvert the systemic glucose metabolism to support growth of leukemia cells. Briefly, leukemia mice are found to have characteristics of both type 2 and type 1 diabetes: insulin resistance and insulin defect, conditions that inhibit glucose utilization in normal tissues. Mechanistically, leukemia induces a high-level production of IGFBP1 from adipose tissue, which results in insulin resistance. Further, leukemic disease impairs gut functions causing loss of gut-derived serotonin and dysbiosis. Serotonin loss results in inhibition of insulin secretion and dysbiosis leads to insulin resistance and less production of microbiota-derived short chain fatty acids such as butyrate and propionate. Supplementation of serotonin or SCFAs impedes leukemia progression. Importantly, combination of serotonin and SCFAs supplementations drastically reduce leukemic burden and prolong survival of leukemic mice by directly increasing the uptake and utilization of glucose in normal tissues. Together, these data demonstrate that restoration of normal glucose regulation may be a feasible strategy to suppress systemic growth of malignant cell types. Taking together, these two studies suggest that interventions by targeting either intracellular metabolism of LSCs or systemic metabolism are effective means for disease management.报告人简要介绍︰叶浩彬,学士,男,1988年落地,结束学业于罗切斯特高校历史高校(University of Rochester Medical Center),现为印第安纳高校军事高校(University of Colorado Medical 坎普us)大学生后,重要举办白血病病理和白血病干细胞代谢及其微情形切磋。已在Cancer Cell、 Cell Stem Cell、blood和Journal of Biological Chemistry等刊物上刊登散文及摘要8篇。担任Cancers、International Journal of Molecular Sciences、Molecules和Vaccines等国际学术期刊审阅稿件人。

报告人:陈嘉乐(酒花之国宇宙航行核心德文 Aerospace Center

个人简单介绍:

附件:

主题材料:基于高分辨质谱本领的针对油砂工业废水的条件化学钻探

刘源,二零一八年6月硕士完成学业于德国Frye贝格地质学院(TU Bergakademie Freiberg),大学生散文德意志联邦共和国评分别获得得“magna cum laude”(卓越),现继续硕士后钻探。专门的学问是布局部质学,研商方向为地质进度的数值模拟,硕士阶段师从国际著名岩石力学和数值模拟专家HeinzKonietzky教师,学习用离散元数值模拟方法化解地质结构进度中的断裂增加难点,属学科交叉商讨。第一遍将离散元(DEM)方法应用到大口径的地质结构演化模拟中,建构了拉分盆地的断裂和盆地演变进程的数值模型,通过对模型尺度和断裂参数的深入分析商讨,第三遍基于离散元方法(颗粒流PFC)模拟了拉分盆地转身一变演变进度中的断裂扩充难点,揭发了菱形拉分盆地的源于难点,给出了总计拉分盆地产生的微小位移和纤维时间的诀要,为拉分盆地的产生演变进度提供了新的探讨思路和办法,相关成果以率先笔者兼通信作者揭橥于组织地质学界超级期刊Tectonics(IF:3.784)上。

报告人:黄荣夫(加拿大阿尔伯塔大学,副商量员)

报告二:Understanding the mechanical properties of shale in nanoscale

标题:生物成因煤层气的探究及商业化

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